By Alan R. Shalita, James Q. Del Rosso, Guy Webster
The American pimples and Rosacea Society is devoted to advancing the technology on the topic of pimples and rosacea and to improving conversation among these drawn to those ailments. this article isn't really intended to be a accomplished treatise on all elements of pimples, yet relatively to handle significant sights by means of said inspiration leaders within the box within the desire that it'll result in additional learn, dialogue, and refinement of our suggestions of either the pathogenesis and the therapy of acne.
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Extra resources for Acne Vulgaris
Cutis 2002; 70:101 105. 3. Plewig G, Fulton JE, Kligman AM. Cellular dynamics of comedo formation in acne vulgaris. Arch Dermatol Forsch 1971; 242:12 29. 4. Vowels BR, Yang S, Leyden JJ. Induction of proinflammatory cytokines by a soluble factor of Propionibacterium acnes: implications for chronic inflammatory acne. Infect Immun 1995; 63:3158 3165. 5. Thiboutot D. Hormones and acne: pathophysiology, clinical evaluation, and thera pies. Semin Cutan Med Surg 2001; 20:144 153. 6. Thiboutot D, Jabara S, McAllister JM, et al.
Arch Dermatol 1982; 118:903 905. 6. Plewig G, Fulton JE, Kligman AM. Cellular dynamics of comedo formation in acne vulgaris. Arch Dermatol Forsch 1971; 242:12 29. 7. Holmes RL, Williams M, Cunliffe WJ. Pilosebaceous duct obstruction and acne. Br J Dermatol 1972; 87:327 332. 8. Knutson DD. Ultrastructural observations in acne vulgaris: the normal sebaceous follicle and acne lesions. J Invest Dermatol 1974; 62:288 307. 9. Toyoda M, Morohashi M. Pathogenesis of acne. Med Electron Microsc 2001; 34(1):29 40.
Further studies have provided evidence that the expression of keratin markers is upregulated by inflammatory cytokines, such as interleukin-1 (IL-1) and IFN-a, and growth factors, including TGF-a and EGF. This provides evidence for the involvement of inflammatory responses in the very early stage of acne lesion development (13 15). Comedogenesis might also be due to reduced desquamation caused by increased cohesion between ductal keratinocytes, or a combination of keratinocyte hyperproliferation and reduced desquamation (6).