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By C. Wüster, T. Cowell

Complement factor: Hormone examine 2000, Vol. fifty four, No. 1

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Extra resources for Analytical Methods in Clinical Osteology: Useful Predictors of Long-Term Outcomes or a Waste of Time and Money? 4th Kigs Kims Expert Meeting on Growth ... Taormina, November (Hormone Research)

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There were no significant differences in the prevalence of fractures in patients with isolated GHD compared with those with additional pituitary hormone deficiencies (table 1). Furthermore, there were no significant differences in the prevalence of fractures in patients receiving glucocorticoid, L-thyroxine or oestrogen replacement therapy compared with patients without these deficiencies (table 2). Diabetes insipidus was associated with a significantly higher fracture rate in men but not in women (men, 35% versus 27%; women, 26% versus 26%).

Disuse-mode remodelling is therefore thought to account for all adult-acquired osteopenias, including those induced by weightlessness [26, 40, 41]. Under higher strains, completed BMUs begin to make and resorb equal GH and Bone Horm Res 2000;54(suppl 1):36–43 37 Fig. 1. Combined modelling and remodelling effects on bone strength and ‘mass’. The horizontal line at the bottom of the figure suggests typical peak bone strains from zero on the left to the fracture strain on the right (Fx), plus the locations of the remodelling, modelling and microdamage strain thresholds (MESy, MESm, MESp, respectively; this article does not discuss microdamage).

Overall, the prevalence of fractures did not differ in the naive and non-naive groups (fig. 3). 6%, p ! 0001). In contrast, obesity (body mass index 127 kg/m2) did not appear to have an effect on fracture rates (obese patients versus non-obese patients, 30% versus 27%). Table 1. 2 Discussion Table 2. 0 nificant difference was found between naive and nonnaive patients in two subgroups: patients over 50 years of age (34% versus 27%, respectively; p ! 05) and men under 30 years of age (32% versus 17%, respectively; p !

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